Electrophysiological Measurement of Noxious-evoked Brain
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For example, through a process of central sensitization, the firing of dorsal horn nociceptors can change dramatically in the setting of injury (produced by either tissue or nerve damage). Secondary are hyperalgesias occurring outside such an area. (2) According to another definition, a hyperalgesia is primary when the reason is sensitization of nociceptors and secondary when it is due to alterations of central synaptic transmission. Secondary hyperalgesia occurs outside the injury site and is characterized by mechanical hyperalgesia only. Hyperalgesia in inflammatory processes corresponds to primary hyperalgesia. Hyperalgesia in referred pain and neuropathic pain resembles secondary hyperalgesia (Table 3).
For practical reasons, the first type of definition should be preferred, since it is purely descriptive. Secondary hyperalgesia occurs outside the injury site and is characterized by mechanical hyperalgesia only. Hyperalgesia in inflammatory processes corresponds to primary hyperalgesia. Hyperalgesia in referred pain and neuropathic pain resembles secondary hyperalgesia (Table 3). Hyperalgesia is divided into two categories: primary and secondary.
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• Spread of symptoms. 3 Nov 2015 SECONDARY HYPERALGESIA • Rational treatment of secondary hyperalgesia is the drug/technique that can prevent the occurrence of Secondary hyperalgesia develops in uninjured tissue surrounding the site of injury, and is thought to be a result of sensitization in the central nervous system, 4 Feb 2015 Secondary hyperalgesia involves the surrounding uninjured area. (Copyright Cleveland Clinic Foundation.) Each sensory cell has specific Secondary hyperalgesia is defined as an increase in pain sensitivity when a noxious stimulus is delivered to a region surrounding, but not including, the zone of injury (increased pain sensitivity outside of the area of injury or inflammation). Secondary hyperalgesia is due to central neuron sensitization and requires continuous nociceptor input from the zone of primary hyperalgesia for its maintenance.
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Before incision, catheterized rats were acclimated to the testing room and testing apparatus (i.e., individual plexiglass compartments measuring 26 × 11 × 20 cm with 12 × 12 mm mesh floors) for 30 min on each of three sequential days. tion, and secondary hyperalgesia surrounding the site of stimulation [4–8]. The area of secondary hyperalgesia is characterised by reduced thresholds for mechanical stimulation, and the size of the area can be quantified by monofilament stimulation.
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Science Publ Hyperalgesia that will not respond to a further dose increase of oxycodone may very rarely depression secondary to oxycodone overdose. HT1-7) [6-9] based on pharmacological properties, second messenger coupling and the 5-HT7R may be involved in pain, hyperalgesia and neurogenic Two of three patients with multiple chemical sensitivity had less symptoms and secondary hyperalgesia after transcranially applied pulsed electromagnetic fields Allodynia, hyperalgesia, dysesthesia, increased wind-up, regional/general pain The secondary purpose was to carry out a quality assurance of record keeping s. k. secondary hospitals.
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77 Baumgärtner U, Magerl W, Klein T, Hopf HC, Treede RD. Secondary hyperalgesia refers to the increase in sensitivity to mechanical nociceptive stimuli delivered outside the area of tissue injury. Previous studies have suggested that secondary hyperalgesia is mediated by a specific class of myelinated nociceptors: slowly adapting A-fibre mechano- and heat-sensitive (AMH) type I nociceptors. lar VR environment on capsaicin-induced ongoing pain and secondary hyperalgesia.